1. Asphyxia
2. Distributive shock (e.g., anaphylaxis)
3. Multi-organ dysfunction syndrome (MODS)
4. Multisystem trauma
5. Sepsis/septic shock
6. Systemic inflammatory response syndrome
(SIRS)
7. Toxic ingestions/inhalations (e.g., drug/alcohol
overdose)
8. Toxin/drug exposur
CCRN Review Questions & More
This BlogSpot was created for any critical care nurse seeking CCRN certification. CCRN is nationally recognized as a validation of excellence. This vision of excellence & focus on quality care is the driving force behind this BlogSpot. It is designed to empower nurses & create a culture of certification. CCRN is a great accomplishment & you can do it, just study!
Sunday, October 23, 2011
Thursday, September 8, 2011
Cardiac Markers
Cardiac markers | |||
Marker | Initial Elevation | Peak Elevation | Return to Baseline |
Myoglobin | 1-4 h | 6-7 h | 18-24 h |
CK-MB | 4-12 h | 10-24 h | 48-72 h |
Cardiac Trop I | 3-12 h | 10-24 h | 3-10 d |
Cardiac Trop T | 3-12 h | 12-48 h | 5-14 d |
The troponin I is the most sensitive cardiac marker, detectable in serum 3-6 hours after an MI, and its level remains elevated for 14 days.
Wednesday, September 7, 2011
Anterior Wall MI
Anterior wall infarction results from occlusion of the proximal LAD artery. ST-segment elevation is expected in leads V1 through V4 on the 12-lead ECG, as shown in. If the left main coronary artery is occluded, the ECG manifestations will involve almost all of the precordial leads V1through V6 and leads I and aVL (see above). These specific groups of ECG changes that help to locate the part of the heart that is infarcting are called indicative changes. A large anterior wall MI may be associated with left ventricular pump failure, cardiogenic shock, or death.
LATERAL WALL INFARCT
Left lateral wall infarction occurs as a result of occlusion of the circumflex coronary artery. On a 12-lead ECG, new Q waves and ST-segment T-wave changes are seen in leads I, aVL, V5, and V6. In reality, very few patients present with only lateral wall ECG changes. Often times some anterior wall leads (V3 and V4) may show evidence of injury or infarction.
ANSWERS #1-20
1. A - Nipride
2. A - PVR
3. A - Pulmonary Edema
4. A - RAP
5. C - Pulmonary HTN
6. C - Vasoconstriction
7. A - Increased HR, contractility, conductivity
8. D - Vasodilation, bronchodilation
9. B - Dilation of the renal and mesenteric arteries
10. D - Phenylephrine
11. C - Dobutamine
12. A - Isoproternol
13. B - Dopamine at 2 mcg
14. B - V3
15. D - Phenylephrine
16. C - Start large bore IVs, get lab samples and fluid bolus
17. C - Pericardial tamponade
18. D - Perform synchronized cardioversion
19. C - Atrial fib
20. C -Bleeding at the groin site
IF ANYONE NEEDS THE RATIONALE FOR A PARTICULAR ANSWER, EMAIL ME!
3. A - Pulmonary Edema
4. A - RAP
5. C - Pulmonary HTN
6. C - Vasoconstriction
7. A - Increased HR, contractility, conductivity
8. D - Vasodilation, bronchodilation
9. B - Dilation of the renal and mesenteric arteries
10. D - Phenylephrine
11. C - Dobutamine
12. A - Isoproternol
13. B - Dopamine at 2 mcg
14. B - V3
15. D - Phenylephrine
16. C - Start large bore IVs, get lab samples and fluid bolus
17. C - Pericardial tamponade
18. D - Perform synchronized cardioversion
19. C - Atrial fib
20. C -Bleeding at the groin site
IF ANYONE NEEDS THE RATIONALE FOR A PARTICULAR ANSWER, EMAIL ME!
Sunday, September 4, 2011
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